Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the other hand, our power to rescue SERT operate in KI midbrain synaptoneurosomes from the inhibition of FAK implies elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by improved pFAK localization in five-HT https://pro33login12222.blogsumer.com/31618065/top-pro33-login-secrets